ALFABolin is an injectable steroid with both anabolic activity. It is available in both oral and injection formats and is marketed under the trade names Prednisone and Levitra. It is manufactured by Hoffman-La-Roche and produces approximately 1 million units per year. ALFABolin and its generic version Lufenuron are available throughout the world. It has been used to treat a range of conditions including joint pain, inflammatory bowel disease, athlete's foot, impetigo, warts, hepatitis, asthma, benign prostatic hyperplasia, low back pain, osteoarthritis, reactive arthritis, and multiple sclerosis (MS) and belongs to the class of hormones that stimulate the body's natural immune system and can reverse or prevent the progression of hormonal imbalance. It works by binding the enzyme citrate lyase and thereby preventing the accumulation of citrate crystals in the synovial fluid near the endodontium and replacing it with similar water soluble substances. ALFABolin is manufactured as a synthetic derivative of the natural compound, 5a-reductase. The major active ingredient, methanolonylmethane, is also a derivative of this chemical. The efficacy and the toxicity of the drug are not known, even though there have been cases of serious liver damage in patients taking high doses of alphabolin. Due to these uncertainties it is recommended that alphabolin not be used for internal uses and that pregnant or nursing women and people under the age of 18 should not take the drug. The use of drug alphabolin for testosterone treatment is accompanied with several side effects such as an increase in blood pressure and heart rate, increase in heart rate and increase in blood pressure in women. Some additional side effects may also be noted such as restlessness, slowed metabolism, swollen limbs, headache and muscle weakness. Normally such side effects subside within a few days and the body adapts to the drug. In some rare cases severe adverse events have been reported such as hyperthyroidism. Long term use of alphabolin is known to cause hypogonadism, which is the opposite of its effect. This condition is characterized by decreased production of testosterone leading to symptoms similar to hypogonadism. Hypogonadism is treated differently than testosterone replacement therapy in that patients are treated with prescription medications and regular dosages of alphabolin are stopped. The possible side effects of long term use of alphabolin are the same as those of long term use of anabolic steroids. In terms of side effects related to testosterone replacement therapy, there are three major ones. First, acne at the face, neck and back can occur. Second, blood in the urine and in semen. Sometimes bleeding of the prostate can occur. Third, enlargement or growth of the prostate gland can occur. Because of these risks and because of the fact that alphabolin is not approved for use in treating gynecomastia, the Food and Drug Administration (FDA) has placed it on the list of drugs that are generally not advised. Gynecomastia, however, does not occur due to alphabolin but rather due to the excessive growth of breast tissue. So alphabolin does not contribute to the development of gynecomastia. If you take alphabolin for cutting cycles in your body, it is important to be aware of what some of the side effects of the drug may be. Firstly, there is the risk of bleeding of the veins and arteries as well as peripheral vascular pathology, which can cause swelling, edema, or infection of the skin. There is also a risk of thromboembolism. Thromboembolism is the possible formation of blood clots that can lead to stroke or heart attack. Because alphabolin is an anabolic steroid and works by increasing the amount of anabolic steroids in the system, a rise in the number of circulating anabolic steroids is expected. A possible complication from the use of alphabolin and its use in cutting cycles is formation of a substance known as lactic acid. Lactate is formed when lactic acid combines with carbon dioxide. Excessive lactic acid build up can cause severe injury to the muscle tissue. Because the muscle tissues are deprived of oxygen, alphabolin causes a state of hyperreflexia, or increased exertion through constriction of the cardiovascular muscles, which can result in cardiac arrest. The combination of alphabolin with methenolone has been found to increase the length of hospital stays, the amount of time that patients remain on oxygen, and the frequency of allergic attacks. When alphabolin injections are stopped, the metabolic pathway of the drug is affected and the cause of kidney failure is more likely. Because of these risks, alphabolin should be used cautiously in the relief of asthma and in the treatment of cystic fibrosis. In any case, alphabolin should be given under close medical supervision. It is not an appropriate replacement for an active substance such as an adrenal steroid.
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